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@WengFlyLab
@yuchiunwang
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Published: our study on a new Drosophila gene Moat shows strong polarity-based junctions allow weak myosin to promote productive apical constriction, leading to ectopic folding of ectodermal anterior midgut (ectoAMG). The ectopic folding requires Baz/Par3. molbiolcell.org/doi/full/10.10…
Our new manuscript demonstrates that modulating the levels of adherens junctions change the level of myosin-mediated apical constriction and redefine the morphogenetic boundaries of epithelial folding. High levels of adherens junctions can promote infolding with low myosin. 🧵1/n
A novel protein Moat prevents ectopic epithelial folding by limiting Bazooka/Par3-dependent adherens junctions biorxiv.org/cgi/content/sh… #bioRxiv
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