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Adropin treatment shows promise to limit post-stroke brain damage in mouse study mbi.ufl.edu/2022/10/28/adr…
Subarachnoid hemorrhage-associated brain injury and neurobehavioral deficits are reversed with synthetic adropin treatment through sustained Ser1179 phosphorylation of endothelial nitric oxide synthase frontiersin.org/articles/10.33…
RT@UFNeuroscience RT@UFMBI RT@UFBrainInjury New paper in the Journal of Neuroinflammation. “Receptor-interacting protein kinase 2 (RIPK2) profoundly contributes to post-stroke neuroinflammation and behavioral deficits with microglia as unique perpetrators” jneuroinflammation.biomedcentral.com/articles/10.11…
New paper from our lab published in the Journal of Neuroinflammation. “Receptor-interacting protein kinase 2 (RIPK2) profoundly contributes to post-stroke neuroinflammation and behavioral deficits with microglia as unique perpetrators” jneuroinflammation.biomedcentral.com/articles/10.11…
New paper from our lab published in Journal of Neuroinflammation. “Receptor-interacting protein kinase 2 (RIPK2) profoundly contributes to post-stroke neuroinflammation and behavioral deficits with microglia as unique perpetrators” jneuroinflammation.biomedcentral.com/articles/10.11…
Congratulations to Dr. Eduardo Candelario-Jalil’s lab on their recent publication. neuroscience.ufl.edu/2023/10/05/con…
Receptor-interacting protein kinase 2 (RIPK2) profoundly contributes to post-stroke neuroinflammation and behavioral deficits with microglia as unique perpetrators dlvr.it/Swr9N6
.@UF neuroscientists report treatment with the peptide adropin shows promise to limit post-stroke brain damage in mouse-model study. Preclinical @StrokeAHA_ASA paper unveils potential neuroprotective role of adropin in aged brain after ischemic #stroke. bit.ly/3gTTw6P
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