Mia Petljak
@MPetljakAssistant Prof @nyugrossman @Perlmutter_CC | alumna @BroadInstitute @VertexPharma @sangerinstitute | Somatic mosaicism in cancer & aging 💻🔬 Opinions my own.
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Excited to share our latest work. We introduce an improved version of NanoSeq, a duplex sequencing protocol with <5 errors per billion bp in single DNA molecules, and use it to study the somatic mutation landscape of oral epithelium in >1000 people. 1/n medrxiv.org/content/10.110…
🌟 Congratulations to HHMI Investigators David Mangelsdorf and David Pellman who have been elected as members of the National Academy of Medicine (@theNAMedicine)! More: hhmi.news/3YywtBS
It was a great pleasure to discuss advances in human genetics & genomics last week in Beijing with great colleagues, fantastic hosts & over the best food, sights and most inspiring research 🧬💕
Excited to see Ben Ebert as the new CEO at DFCI! His brilliance is matched only by his compassion and humanity. Ben’s been a fantastic collaborator and advisor to so many of us. Looking forward to witnessing all that’s ahead under his leadership.
Dr. Laurie Glimcher, CEO of Dana-Farber Cancer Institute, said she would step down next month. Dr. Benjamin Ebert, chair of Dana-Farber’s medical oncology department, will take over on Oct. 1. trib.al/WVBqnUH
Wow!
Exciting new inroads into human biology and therapy development: Human intestinal organoids with an autologous tissue-resident immune compartment! #Gjorevski #Cabon #IHB @Nature @IHB_Research nature.com/articles/s4158…
Paper out!📢 @AnupriyaSingha9 and #HannahStyers from the lab find acute resistance to targeting KRAS in pancreas cancer is surprisingly driven by a classical epithelial state. Co-targeting classical cells and KRAS augments response - clinically important! aacrjournals.org/cancerdiscover…
Now out @Nature, our paper on HiDEF-seq: single-molecule DNA sequencing that resolves single-strand changes (mismatches and damage)! go.nature.com/3KG7Njh
Clustered mutations occur more frequently than expected, and translesion synthesis (TLS) drives collateral mutagenesis on both the leading and lagging strands. SNVs are clustered downstream of deletions (biased towards deletion of T) but upstream of insertions. [4/13]
"Tempora mutantur, et nos mutamur in illis". Ovid knew long ago that our somatic genomes evolve throughout life... Super excited to share our new paper on connecting clonal outgrowth in somatic evolution with epigenetic profiles @Nature Open here: rdcu.be/dHkeS 🪡
I am thrilled that our paper on kidney cancer is now out in @Nature!!! nature.com/articles/s4158…
Pop it in your diary for next week!
Our next Early Career Immunology seminar is Carla Nowosad @CarlaTheNove @nyuniversity Immunoreceptors in the intestinal microenvironment Tues April 30, PST 9am Zoom temple.zoom.us/webinar/regist…
Big news, Nowosad Lab is hiring a technician! Come and help us figure out how B cells tell friend from foe in the intestine. You can message me, check out our lab website, or apply here > jobs.nyulangone.org/job/20205972/r…
Interested in learning more about the many functions of Reactive Oxygen Species in cancer? Check out our recent review in @NatureCancer Special thanks to Kat who in collaboration with Ahmed and @sayinlab put together this comprehensive review article nature.com/articles/s4301…
Very excited to share our latest preprint on somatic mutations in the normal stomach. We found many surprises, incl. hypermutant glands, recurrent somatic trisomies and a unique landscape of driver mutations. Thread below! biorxiv.org/content/10.110…
Children who develop two independent tumors during childhood are extremely rare. We wanted to shed light into those exceptional cases using genome analysis We also reveal the mutational footprint of chemotherapy in normal tissues using duplex sequencing of autopsy samples 👇
Our work on the origins of second malignancies in children is out in Cancer Discovery @IRBBarcelona @bbglab @IRSJD_info doi.org/10.1158/2159-8… 1/16
Now online! APOBEC3A and APOBEC3B drive distinct mutational landscapes in tumors. We showed how A3A and A3B substrate preferences for different hairpin structures can be exploited to identify patients' tumors with mutations caused by either A3A or A3B. nature.com/articles/s4146…
Odličan primjer su prilike naše mlade znanstvenice Mije Petljak @MPetljak koja je nedavno otvorila svoj lab na NYU Grossman Medical School i traži postdoktoranda, senior znanstvenika, i čak remote interne iz područja bionformatike! #internships #postdocs #jobopportunity
How did we lose our tail? A simple question.. but it wasn't really asked before! We discovered a plausible scenario for the genetic mechanism that led to tail loss. Amazing that such a big change may have been caused by such a small genetic event. nature.com/articles/s4158… @BoXia7
![Director, Computational Biology. [nrobine]@nygenome[.org] RNA-Seq / Cancer Genomics / anything NYC / mostly in English, sometimes in French.](https://pbs.twimg.com/profile_images/1455189540335931393/YncNZBxK.jpg)
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